Researchers from Helmholtz Munich and LMU have identified a mechanism that may explain the neurological symptoms of Long COVID. The study, published in the journal Cell Host & Microbe under the title “Persistence of spike protein at the skull meningis-brain axis may contribute to the neurological sequelae of COVID-19,” reveals that the characteristic spike protein of the COVID-19 pathogen remains detectable within the skull long after infection. This persistent accumulation of the spike protein could contribute to the neurological long-term effects of COVID-19. The findings of the study open new avenues for the diagnosis and treatment of Long COVID. Tissue samples and the methodological expertise of NATON were utilized for the research.
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